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CCR9-CCL25 interactions promote cisplatin resistance in breast cancer cell through Akt activation in a PI3K-dependent and FAK-independent fashion

机译：CCR9-CCL25相互作用通过PI3K依赖性和FAK依赖性的Akt激活促进乳腺癌细胞的顺铂耐药性

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Johnson-Holiday, Crystal; Singh, Rajesh; Johnson, Erica L; Grizzle, William E; Lillard, James W; Singh, Shailesh;
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年度 2011
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正文语种 {"code":"en","name":"English","id":9}
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1. CCR9-CCL25 interactions promote cisplatin resistance in breast cancer cell through Akt activation in a PI3K-dependent and FAK-independent fashion [J] . Crystal Johnson-Holiday, Rajesh Singh, Erica L Johnson, World Journal of Surgical Oncology . 2011,第1期

机译：CCR9-CCL25相互作用通过PI3K依赖性和FAK依赖性的Akt激活促进乳腺癌细胞的顺铂耐药性
2. CCR9 interactions support ovarian cancer cell survival and resistance to cisplatin-induced apoptosis in a PI3K-dependent and FAK-independent fashion [J] . Erica L Johnson, Rajesh Singh, Crystal M Johnson-Holiday, Journal of Ovarian Research . 2010,第1期

机译：CCR9相互作用以PI3K依赖性和FAK依赖性方式支持卵巢癌细胞的存活和对顺铂诱导的凋亡的抵抗
3. CCR9-CCL25 interaction suppresses apoptosis of lung cancer cells by activating the PI3K/Akt pathway [J] . Li Baijun, Wang Zhiwei, Zhong Yonglong, Medical oncology . 2015,第3期

机译：CCR9-CCL25相互作用通过激活PI3K / Akt途径抑制肺癌细胞的凋亡
4. Breast Cancer Cells Response to the Antineoplastic Agents Cisplatin, Carboplatin, and Doxorubicin at the mRNA Expression Levels of Distinct Apoptosis-Related Genes, Including the New Member, BCL2L12 [C] . HELLINIDA THOMADAKI, ANDREAS SCORILAS Meeting on Cell Signaling World: Signal Transduction Pathways as Therapeutic Targets . 2007

机译：在不同凋亡相关基因（包括新成员BCL2L12）的mRNA表达水平上，乳腺癌细胞对抗肿瘤药顺铂，卡铂和阿霉素的反应
5. Sensitivity of MCF-7 breast cancer cells to anticancer drugs is decreased by activation of the PI3K/PDK/Akt signal transduction pathway. [D] . Navolanic, Patrick M. 2004

机译：通过激活PI3K / PDK / Akt信号转导途径，MCF-7乳腺癌细胞对抗癌药物的敏感性降低。
6. CCR9-CCL25 interactions promote cisplatin resistance in breast cancer cell through Akt activation in a PI3K-dependent and FAK-independent fashion [O] . Crystal Johnson-Holiday, Rajesh Singh, Erica L Johnson, 2011

机译：CCR9-CCL25相互作用通过PI3K依赖性和FAK依赖性的Akt激活促进乳腺癌细胞的顺铂耐药性
7. CCR9-CCL25 interactions promote cisplatin resistance in breast cancer cell through Akt activation in a PI3K-dependent and FAK-independent fashion [O] . Crystal Johnson-Holiday, Rajesh Singh, Erica L Johnson, 2011

机译：CCR9-CCL25相互作用通过PI3K依赖性和FAK依赖性的Akt激活促进乳腺癌细胞的顺铂耐药性

CCR9-CCL25 interactions promote cisplatin resistance in breast cancer cell through Akt activation in a PI3K-dependent and FAK-independent fashion

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